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328  Corrosive Ingestion and Oesophageal Replacement


                                                  Key Summary Points
            1.  A preventive scheme is essential to avoid the devastating   5.  Fever, shock, dyspnoea, and acute abdomen strongly indicate
              accidental injuries due to corrosive ingestion.     oesophageal or gastric perforation.
            2.  Alkaline injury is more extensive than acid injury due to the   6.  Dysphagia is the late presentation due to stricture.
              delay in the protective reflexes in an acid injury and rapid   7.  Diagnosis is by imaging and endoscopy.
              penetration of alkali into the tissues.
                                                               8.  The goals of therapy are to prevent and treat perforation as
            3.  The most severe caustic injury generally occurs in   early as possible, to avoid strictures of the oesophagus and
              the narrowest portion of the oesophagus, usually the   stomach, and to replace or bypass the damaged organ to allow
              midoesophagus in the region of the aortic arch.     normal swallowing of food.
            4.  Early clinical manifestations include persistent salivation,   9.  Therapy is variable.
              dysphagia, hoarseness of voice and stridor, retrosternal chest
              pain, and hematemesis.





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